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#1
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So antipsychotic treatments, when properly managed are unlikely to result in EPS?
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC3018852/ | ||
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#2
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Quote:
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#3
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Dystonia is an acute, alarming involuntary movement disorder that can be painful and distressing, and erodes patient trust and compliance. 31 It is characterized by briefly sustained or intermittent spasms or contractions of antagonistic muscle groups resulting in twisting and repetitive movements or postures. Drug-induced dystonia can affect any muscle group, but most commonly involves the head, neck, jaw, eyes and mouth resulting in spasmodic torticollis, retro- or anterocollis, trismus and dental trauma, forced jaw-opening or dislocation, grimacing, blepharospasm, tongue biting, protrusion or twisting, and distortion of the lips.32-34 It is not action or sensory stimulus dependent. More subtle signs, including muscle cramps or tightness of the jaw and tongue with difficulty speaking or chewing, may precede dystonia or occur alone.
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#4
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Parkinsonism
Drug-induced parkinsonism is a subacute syndrome that mimics Parkinson’s disease. Though less alarming than dystonia, it is more common, more difficult to treat and can be the cause of significant disability during maintenance treatment especially in the elderly. Bradykinesia is accompanied by masked facies, reduced arm swing, slowed initiation of activities, soft speech and flexed posture. 32 Bilateral and symmetrical rigidity of neck, trunk and extremities appears with cog-wheeling. Resting or action tremors are also observed symmetrically and can be generalized or take the form of a focal peri-oral tremor (rabbit syndrome). Patients may also experience sialorrhea, and postural or gait disturbances. | ||
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#5
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Any other exaggerated concerns that don’t actually apply the the majority of people on these meds?
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#6
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Akathisia may begin within several days after treatment but usually increases with duration of treatment, occurring in up to 50% of cases within one month and 90% of cases within three months. 32 Akathisia should resolve after drug discontinuation, but could temporarily worsen or persist in withdrawal or tardive forms. | |||
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#7
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Although dopamine receptor blockade occurs within a few hours after administration of antipsychotic drugs, the onset of parkinsonism may be delayed from days to weeks, with 50% to 75% of cases occurring within one month and 90% within three months.
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#8
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Also their doctors are conducting monthly follow ups and monitoring for side effects like these. They are not going to continue to knowingly prescribe something giving someone painful, impeding, or uncomfortable side effects Psychiatrists are MEDICAL doctors. They take a Hippocratic oath, same as the medical field | |||
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#9
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Quote:
i·at·ro·gen·ic /īˌatrəˈjenik/ adjective relating to illness caused by medical examination or treatment. "drugs may cause side effects which can lead to iatrogenic disease" | |||
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#10
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In contrast to acute EPS, TD is insidious in onset, arises only after prolonged treatment and is often masked by ongoing treatment. In addition, TD is irreversible in most cases but usually mild, whereas acute EPS are transient but unmistakable and incapacitating. Even so, TD can become socially disfiguring and severe enough to compromise eating, speaking, breathing or ambulation.
TD presents as a polymorphous involuntary movement disorder.32, 87, 88 In its most common form, TD is characterized by involuntary, non-rhythmic, repetitive, purposeless hyperkinetic movements. Most often, TD affects orofacial and lingual musculature (“buccolinguomasticatory syndrome”) with chewing or bruxism of the jaw, protrusion, curling or twisting of the tongue, lip smacking, puckering, sucking and pursing, retraction, grimacing or bridling of the mouth, bulging of the cheeks, or eye blinking and blepharospasm. Choreoathetoid movements of the fingers, hands, upper or lower extremities are common. Axial symptoms affecting the neck, shoulders, spine or pelvis may be observed. Dyskinesias can affect breathing, swallowing or speech. | ||
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