Covid Spike Proteins (mRNA Vaccines) cause Vascular Damage

[FatherSioux]

You guys ever see those things people stick in their penis? Spike type things? So weird.

[Jibartik]

Darkelf bloodcells can create their own spike protines sorry you tunare loving cuckelves cant.

[usernamebob27]

what about a wood elf with prenerf guise?

[Jibartik]

Masks are OK

Better not let that illusion fade below your nose though.

[usernamebob27]

I guess a wood elf rogue or bard with two illusion masks is probably 50% move immune then just having one of them

[Jibartik]

Guise of the Deceiver

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Mask of Deception

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[Botten]

Originally Posted by usernamebob27 [You must be logged in to view images. Log in or Register.]

It's actually worse then you think...

https://www.youtube.com/watch?v=BNyAovuUxro

The people that get vaccinated, their immune system is now permanently programed to make antibodies that will only detect the spike protein on covid19 in its current form. It doesn't give protection from the entire genetic make up of the viruses. So when the virus eventually mutates a change in the proteins in the spikes the people that were inoculated with the RNA vaccines will created those pre programed antibodies that will literally do nothing.

So you don't get the Flu vaccine every year for the same reason. *roll eyes

[Cecily]

Originally Posted by Jimjam [You must be logged in to view images. Log in or Register.]

Surely then, it would be like the Spanish Flu, where people with the 'best' immune systems were worst affected?

Not necessarily. Covid inhibits type 1 interferon response and that's already impaired in the elderly.

https://www.nature.com/articles/s41591-020-01202-8

"A key feature of coronaviruses (MERS-CoV, SARS-CoV) is a capability to inhibit and delay the type I IFN response, leading to increased viral replication and severe immunopathology. Also, SARS-CoV-2 is able to inhibit the type I IFN responses in infected cells, leading to delayed or overall suppressed type I IFN responses49,50. This allows the virus to replicate and induce more tissue damage, and triggers a more exuberant immune response as the immune system struggles to limit viral replication and to manage dying and dead cells. Immune pathology continues as inflammatory cells flow into the lung and produce large amounts of proinflammatory cytokines, further escalating the situation (Fig. 1c). Such imbalanced immune responses, caused in part by the impaired early type I IFN responses, are the most likely determinant of the overall severity of acute COVID-19 (refs. 50,51,52,53). This is further emphasized by recent results from the COVID Human Genetic Effort54 (https://www.covidhge.com/), which found that inborn errors in the type I IFN pathway55, or the presence of neutralizing autoantibodies to type I IFNs56, were strongly over-represented among individuals who developed life-threatening COVID-19. Whether imbalanced or impaired innate responses also contribute to the development of other disease manifestations such as MIS-C and long COVID remains to be determined."

[usernamebob27]

Maybe watch the video first. The Dutch guy being interviewed is a Doctor of Veterinary Medicine who has specialist expertise in virology and vaccinology.

If he's wrong that's a good thing. If he's right, then things are going to be not so good.

https://www.youtube.com/watch?v=BNyAovuUxro

[HalflingSpergand]

Originally Posted by Cecily [You must be logged in to view images. Log in or Register.]

Not necessarily. Covid inhibits type 1 interferon response and that's already impaired in the elderly.

https://www.nature.com/articles/s41591-020-01202-8

"A key feature of coronaviruses (MERS-CoV, SARS-CoV) is a capability to inhibit and delay the type I IFN response, leading to increased viral replication and severe immunopathology. Also, SARS-CoV-2 is able to inhibit the type I IFN responses in infected cells, leading to delayed or overall suppressed type I IFN responses49,50. This allows the virus to replicate and induce more tissue damage, and triggers a more exuberant immune response as the immune system struggles to limit viral replication and to manage dying and dead cells. Immune pathology continues as inflammatory cells flow into the lung and produce large amounts of proinflammatory cytokines, further escalating the situation (Fig. 1c). Such imbalanced immune responses, caused in part by the impaired early type I IFN responses, are the most likely determinant of the overall severity of acute COVID-19 (refs. 50,51,52,53). This is further emphasized by recent results from the COVID Human Genetic Effort54 (https://www.covidhge.com/), which found that inborn errors in the type I IFN pathway55, or the presence of neutralizing autoantibodies to type I IFNs56, were strongly over-represented among individuals who developed life-threatening COVID-19. Whether imbalanced or impaired innate responses also contribute to the development of other disease manifestations such as MIS-C and long COVID remains to be determined."

Stop regurgitarding you guys don't know shit